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Stories from the dialysis comunity across the globe.
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Niacin may help the body fight against Staph. aureus infection. via CEBPE gene. |
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EurekAlert: In laboratory tests with mice and human blood, Cedars-Sinai scientists found that vitamin B3 increased by up to 1,000 fold the ability of the immune system to kill staph bacteria. Beyond its findings related to vitamin B3, the study indicates that similar targeting of the CEBPE gene with other compounds may offer a new immune-boosting strategy to fight bacterial infections.
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Dr. John Stokes, University of Iowa Division of Nephrology Chief, in Memoriam. |
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University of Iowa: A memorial service for Dr Stokes will be held on Saturday, Sept. 1, 2012 at St. Andrews Presbyterian Church in Iowa City (1300 Melrose Avenue) beginning at 9:00 am. Letters of sympathy can be sent to his wife Jackie. For contact information, please contact the University of Iowa.
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Just in time sequence analysis help solve how a multidrug resistant Klebsiella spread. |
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NY Times: Dr. Segre, a genome researcher, proposed sequencing the entire genome of the first patient’s bacteria and comparing it with the genome sequences of bacteria from other infected patients. That could enable scientists to detect minute genetic changes that were the bacterium’s fingerprints. And they could use that knowledge to track the chain of infection.
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CD4 T cells linked to immunologically-mediated atherosclerosis; vaccine may be possible. |
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EurekAlert: Dr. Ley said he believes the antigen involved is actually a normal protein that the body mistakes as being foreign and therefore launches an immune attack resulting in inflammation in the arteries. "Essentially, we're saying that there appears to be a strong autoimmune component in heart disease," he said, explaining that autoimmune diseases result from the body's mistaken attack on normal cells. "Consequently, we could explore creating a "tolerogenic" vaccine, such as those now being explored in diabetes, which could induce tolerance by the body of this self-protein to stop the inflammatory attack."
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(+)-naloxone blocks opioid addiction by an immunologic mechanism. |
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EurekAlert: IMAGE: This is concept art depicting addiction by Joshua Burton (School of Medical Sciences, University of Adelaide). Click here for more information. "The drug (+)-naloxone automatically shuts down the addiction. It shuts down the need to take opioids, it cuts out behaviours associated with addiction, and the neurochemistry in the brain changes – dopamine, which is the chemical important for providing that sense of 'reward' from the drug, is no longer produced."
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